Upregulation of vimentin and reorganization of podocyte IFs have also previously been observed in rats with puromycin aminonucleoside nephrosis [39], [40], mice with podocyte-selective deletion of the microRNA generating enzyme, dicer, which results in podocyte foot process effacement, split GBMs, and proteinuria [41], and in human glomerular diseases [42], [43]. Here, VIM is linked to glomerular disorder.