IL2 and T-cell non-Hodgkin lymphoma: Moreover, because they chose to work with continuously proliferating T cell lymphomas and hybridomas instead of using normal IL-2-dependent T cell clones, they could not examine the function of the TCR complex in T cell activation, proliferation and any differentiated function such as cytolysis, beyond showing that their MoAbs interfered with antigen-induced IL-2 production.