Bcl signaling is affected by arresten in both endothelial cells and, according to our current data, also in carcinoma cells (Figure 5G–H and [18], [19]); the expression of anti-apoptotic Bcl-xL decreased in both cell types, but the amount of pro-apoptotic Bax increased only in the Arr-HSC carcinoma cells. This evidence concerns the gene BAX and carcinoma.