Atherosclerosis and NAFLD progression are both accompanied by inflammation development which involves several factors, including various chemokines (monocyte chemoattractant protein 1; MCP-1), cytokines (tumor necrosis factor-α; TNF-α), interleukin-6 (IL-6) and C-reactive protein (CRP) (3–6). This evidence concerns the gene CCL2 and metabolic dysfunction-associated steatotic liver disease.