Similar compensatory mechanisms also may play a role in TLR2-, NOD2- and FPR1-deficient mice as these mice also eventually clear the infection and the cellular composition of 7/4+ neutrophils and MOMA2+ monocytes/macrophages on day 10 after infection in TLR2-, NOD2-, FPR1-deficient mice was similar to the cellular composition in wt mice whereas IL-1β-deficient mice had a paucity of 7/4+ neutrophils at this time point (Fig. S16). The gene discussed is FPR1; the disease is infection.