Recently, we showed that while some genes are specifically and differentially expressed between PCa harboring ERG or ETV1 rearrangements, others are commonly dysregulated between these tumor molecular subtypes and PCa without ETS rearrangements, with ETV4 and ETV5 positive tumors clustering together with those with ETV1 rearrangement [33]. This evidence concerns the gene ETV1 and posterior cortical atrophy.