This conclusion was also recently corroborated by a report on the phenotype of myostatin-null heterozygote GRMD dogs, showing that muscle hypertrophy is deleterious in this model of DMD (J.N. Kornegay, oral communication at 4th Annual Symposium of the ESVN Neurological Genetic Diseases Trier, 23–24 september 2011). This evidence concerns the gene MSTN and Duchenne muscular dystrophy.