CFTR and cystic fibrosis: A similar effect (reduced IGA in CF cells) was observed using IB3-1 and S9 cells (the last are wt-CFTR complemented IB3-1 cells), even though CFTR-stimulation instead of its pharmacological inhibition was used in this case, effects corresponding to different molecular mechanisms (PKA phosphorylation of the CFTR domain R vs. blocking of the channel transport activity).