The fact that samples carrying genotypes associated to lower risk also displayed reduced expression of several other chemokines and receptors, as well as with reduced intensity of the myocardial infiltrate, may suggest that the CXCL9 and CXCL10 genotypes control the intensity of myocarditis through modulation of CXCL9 and/or CXCL10 production and its ensuing effects on chemokine ligands of the CXCR3/CCR5/CCR4 receptors. This evidence concerns the gene CXCR3 and myocarditis.