Interestingly, intermittent docetaxel treatment of ovarian tumours led to significant upregulation of ABCB1, ABCC10, bcl2 and TUBB3 among other genes involved in drug resistance whereas continuous docetaxel did not induce upregulation of any analysed genes but instead led to a downregulation of ABCC10, TUBB3 and stathmin [19]. The gene discussed is TUBB3; the disease is ovarian neoplasm.