In humans, patients with acute myocardial infarction who received an intracoronary infusion of bone marrow derived progenitors (sorted for markers CD34/CD45 and CD133) or peripheral blood-derived progenitors (plated for 3 days and positive for endothelial markers such as CD31, KDR (VEGFR2), von Willebrand factor and CD105 as well as uptake of low density lipoprotein and lectin binding) saw a beneficial effect in post-infarction remodeling processes, such as a global increase in ejection fraction and a decrease in infarct size [11], [12]. The gene discussed is KDR; the disease is acute myocardial infarction.