Previous studies in humans with decompensated LVH have shown a decrease in the relative density of connexin 43 gap junctions at the intercalated disc.39 Our studies demonstrate that the density of connexin 43 gap junctions at the intercalated disc in individuals with LVH+SCD and in individuals with LVH + unnatural death (Control Group A) is similar. Here, GJA1 is linked to Schnyder corneal dystrophy.