In a further study, the same research group, utilizing a radioligand-binding assay, confirmed that Abs to IFN-ω occur in the totality of APS-1 patients and beat the prevalence of Abs to two subtypes of IFN-α, namely IFN-α2 and IFN-α8, that in turn are found in a high percentage of patients with thymoma-associated myasthenia gravis [84]. This evidence concerns the gene IFNA2 and autoimmune polyendocrine syndrome type 1.