Remarkably, the binding of PRC1 hinders the access of other chromatin remodeling complexes such as SWI/SNF that may have transcription-enhancing functions[23], which implies that out of the antagonistic partners of SYT-SSX in synovial sarcoma, CpG may ultimately dominate over SWI/SNF[10]. The gene discussed is SMARCA1; the disease is synovial sarcoma.