For example, injection of apoptotic bodies has been shown to prevent the development of experimental immune complex-mediated inflammatory arthritis [66], accelerate the resolution of acute inflammation [8], expand a pool of TGF-β tolerogenic T cells [67], non-specifically facilitate allogenic bone marrow engraftment [68], and interfere with graft rejection and the development of graft-versus-host disease [69]. The gene discussed is TGFB1; the disease is graft versus host disease.