SLC4A11 and Fuchs endothelial corneal dystrophy: A role for SLC4A11 has been suggested via a small number of late-onset FECD patients carrying coding-sequence mutations within samples of 89 mainly Chinese late-onset FECD patients [38], and 189 Caucasian nuclear families with late-onset FECD [27], with no apparent independent effect on CCT, although association was not established in these studies.