Secondary hyperparathyroidism in uremia (s-HPT)—a disorder caused by progressive loss of kidney function, low levels of active vitamin D (1,25(OH)2D (calcitriol)), increased phosphate retention, and low levels of plasma ionized calcium (Ca2+) [1, 2]—results in the highly elevated synthesis and secretion of parathyroid hormone (PTH) and enlargement of the parathyroid glands in order to maintain normal plasma Ca2+ and phosphate levels. This evidence concerns the gene PTH and secondary hyperparathyroidism.