We observed that treatment with WDL downregulates PKCε in a dose-dependent manner, but does not decrease phosphorylation of Akt in the same experimental conditions (Fig. 6A and B) which suggests that WDL induces apoptosis in prostate cancer cells via downregulation of PKCε but not via inhibition of Akt. The gene discussed is AKT1; the disease is prostate carcinoma.