TPSAB1 and infection: Thus, Tps1 control of NMR and CCR could provide a mechanistic framework for understanding how virulence genes are expressed early in infection (when the fungus might be in a glucose-rich, nitrogen-poor environment such as might be found in the host apoplast), and how genes for utilizing alternative carbon sources are derepressed later in infection (when the fungus might be in a glucose-poor environment as colonized cells expire and necrotrophy commences).