CD8A and infection: Although we detect minimal differences in the expression of MHC I molecules and conventional accessory molecules (e.g. co-stimulatory ligands CD80/86) between GFP+ and GFP− neutrophils (data not shown), we cannot formally exclude the possibility of an infection induced alteration in expression of cell surface or soluble molecules in the infected cells which accounts for efficient triggering of effector CD8+ T cells.