SKIC2 and myeloid sarcoma: Our findings reinforce the hypothesis of a possible involvement of these viruses in MS pathogenesis, with the possibility for MSRV of a direct role of effector of pathogenicity, and for EBV of an initial trigger of future MS, years later, since the possibility of the activation of the immuno- and neuro-pathogenic potential of HERV-W by superinfecting EBV is concrete, both in blood and in brain.