DST and pulmonary fibrosis: Different from other findings in which the results of gene expression in pulmonary fibrosis were resulted from the comparisons of the gene expression datasets between fibrous tissues and healthy lung tissues[13] or hypersensitivity pneumonitis lung tissues[15], we found that gene members of the structural constituent of cytoskeleton (keratin, claudin, dystonin) annotation cluster were overexpressed in CPFE fibrotic lesions versus emphysematous lesions.