Genotyping allowed the identification of three subtypes: HNF1α-inactivated HCA (35%–50% of cases), β-catenin-activated HCA (15%–18% of cases), and inflammatory HCA (40%–55% of cases), which could be identified by immunohistochemistry on paraffin-embedded materials [2–6]. Here, HNF1A is linked to hepatocellular adenoma.