AMOT and neoplasm: Further support for the possible dormant phenotype of tumors generated by Clone #1 were the concomitant overexpression of genes such as angiomotin, IGFBP5, and TGF-β2 – which were previously shown to be associated with tumor dormancy – together with down-regulation of CD73, ESM-1, and EGFR – genes associated with rapid tumor growth.