In contrast, less accumulation has been found in camalexin-deficient mutants, resulting in more susceptibility to fungus and bacterial disease compared with wild-type plants, such as cyp79B2/cyp79B3 double mutants, pad3-1 and cyp71A13 mutants were susceptible to A. brassicicola, camalexin-deficient pad4 and pad2 had enhanced susceptibility to P. syringae[18], [36]. This evidence concerns the gene PADI2 and bacterial infectious disease.