However, a higher prevalence of viral myocarditis can be revealed by the application of a comprehensive combination of molecular pathological analysis for the detection of enterovirus RNA from frozen heart tissues, and immunohistochemical techniques for the detection of the enteroviral capsid protein VP1 and both CD3-T-lymphocytes and CD19-B-lymphocytes from formalin-fixed, paraffin-embedded heart tissue. Here, CD19 is linked to viral myocarditis.