Because PG [17], endothelial NOS (e-NOS), and nitric oxide (NO) (but not inducible nitric oxide synthase (i-NOS)), derived (NO)) are crucial for gastric ulcer healing, we compared the status of the mucosal i-NOS/e-NOS ratio as well as the NO and PGE2 levels in the ulcerated and GAE-treated mice. The gene discussed is NOS3; the disease is gastric ulcer.