The absence of double stained cells allowed to formally exclude that the hypointense signals surrounding the tumor were due to cells that, after unspecifically engulfing the Nilo2-mGNPs, migrated to the damage site (Figure 7K–O), in full agreement with the experiments using hp53 or CD3ε control antibodies coupled to the mGNPs (see above). Here, CD3E is linked to neoplasm.