Furthermore, using an antibody cocktail against marker proteins of mitochondrial oxidative phosphorylation (OXPHOS) complex I-V (Fig. 2B), we found that sepsis reduced ∼80% tyrosine phosphorylation of NDUF88 in complex I, succinate dehydrogenase complex, subunit B (SDHB) in complex II, and ubiquinol-cytochrome c reductase core protein II (UQCRC2) in complex III. The gene discussed is UQCRC2; the disease is Sepsis.