BST1 and ovarian cancer: Using a co-culture system to model aspects of the metastatic process occurring in vivo[17], we demonstrated that the extent of transmesothelial migration achieved by each EOC cell line correlates with the level of CD157 and is influenced by the activation of MMPs, further implicating CD157 in the control of a crucial step of ovarian cancer metastasis, that is, mesothelial invasion.