In addition, because neutropenic patients with gastrointestinal tract-derived hepatosplenic candidiasis often develop worsening liver and splenic abscesses immediately after neutrophil recovery [7], the role of Ccr1 in mediating immunopathology in that setting should be investigated using a neutropenic mouse model of gastrointestinal tract-derived invasive candidiasis [50], in which it would be important to determine whether neutrophils that repopulate the bloodstream after recovery from neutropenia express Ccr1. The gene discussed is CCR1; the disease is splenic abscess.