GALNT3 and hyperphosphatemia: The availability of a Galnt3 null mouse, which was not available at the commencement of our study, would further facilitate investigations of phosphate homeostasis, and we therefore pursued studies to establish such a mouse model by assessing the progeny of mice treated with the chemical mutagen N-ethyl-N-nitrosourea (ENU) [32] for the occurrence of soft tissue (ectopic) calcification in association with hyperphosphataemia.