In this largest prospective study of malaria outcomes according to host adhesion receptors, limitations nevertheless included: not characterizing parasite-encoded adhesins or performing adhesion assays with patient-derived iRBC isolates (Ochola et al, 2011); inability to directly measure endothelial ligand expression; and not having quantified ICAM1 and CD36 expression through illness and recovery. This evidence concerns the gene CD36 and malaria.