ERBIN and 22q11.2 deletion syndrome: Discoveries of various Erbin binding partners by later investigations indicate the functional roles of Erbin in determining cell polarity and cell adhesion [7]–[10], since the binding partners of Erbin are mainly the proteins that are the components of adherens junctions, such as p120 catenin family proteins p0071 and δ-catenin, plakophilin-related armadillo-repeat protein-interacting protein, armadillo repeat gene deleted in velocardiofacial syndrome, and the proteins involved in cell attachment to substrates, such as β4-integrin and bullous pemphigoid antigen 1 [5], [8]–[11].