So far, previous studies were mainly focused on the association of OX40L polymorphisms with autoimmune diseases and inflammatory diseases such as systemic lupus erythematosus (SLE), systemic sclerosis (SSc) and myocardial infarction (MI) [14], [15], [16], [17], [18], [19]. This evidence concerns the gene TNFSF4 and myocardial infarction.