MAVS and viral infectious disease: Interestingly, MAVS tyrosine phosphorylation triggered by viral infection began to decrease to a level lower than the uninfected cells beyond 120 min post-infection in HEK293T and MCF-7 cells, while MAVS tyrosine phosphorylation decreased to a level similar to the uninfected cells from 60 min after VSV infection in HepG2 cells, indicating that MAVS phosphorylation kinetics are cell type specific.