These authors reported that the expression of c-KIT and a better prognosis is an “unexpected” finding since, in several tumor types, c-KIT is associated with malignancy [9] as a result of constitutively turned on tyrosine kinase and, in the case of both expression of c-KIT and its ligand SCF, as a result of self-support from an autocrine tumor growth feedback loop [9,19]. This evidence concerns the gene KIT and neoplasm.