In a separate study, mice with keratinocyte- or lung-specific overexpression of TSLP were shown to have an atopic dermatitis- or asthma-like phenotype, characterized by eosinophilia, while mice lacking the TSLP receptor have considerably attenuated disease, lacking eosinophilia [26]. The gene discussed is TSLP; the disease is Increased total eosinophil count.