Confirming the role of anti-eTG in the pathogenesis of DH, Zone et al. demonstrated that passive transfer of both goat IgG anti-eTG and human IgA anti eTG antibodies in SCID mice bearing human skin grafts induces granular IgA deposition in dermal papillae of human skin in a pattern similar to that of DH. The gene discussed is CD79A; the disease is dermatitis herpetiformis, familial.