A great number of STLs possess considerable anti-inflammatory activity related to the inhibition of the transcriptor factor NF-κB [11] that plays a pivotal role in the regulation of the cell homeostasis, apoptosis and tumour growth [12] and it was straightforward to assume a link between the known cytotoxic activity of STLs and their NF-κB inhibitory activity. This evidence concerns the gene NFKB1 and neoplasm.