Greater amounts of IL-1α, IL-2R, IL-12, IL-18, CXCL1, CXCL9, MIF, β-NGF, HGF, C-peptide, GIP, insulin, PAI-1, adiponectin, leptin, resistin and adipsin and lower of ghrelin were secreted by LC patients respect to controls (Figures 1–2). The gene discussed is CXCL9; the disease is laryngotracheoesophageal cleft.