HF-related changes in myocardial gene expression have also been found at various points in the FA uptake and oxidation pathways in humans and animal models for the following genes: Cluster-of-Differentiation 36 (CD36, also known as Fatty-Acid Translocase (FAT) and an important FA transporter across the plasma and outer mitochondrial membranes) [5], [17]; Cardiac Carnitine Palmitoyl-Transferase-1 (CPT-1, the other principal outer mitochondrial membrane FA transporter) [15], [18], [19]; and Long-Chain Acyl-CoA Dehydrogenase (LCAD, a key enzyme of β-oxidation) [18], [20]–[22]. The gene discussed is CD36; the disease is hydrops fetalis.