For example, Suda et al. [42] found that relative expression levels of EFNB3, PLXNA4 and ROBO2 were significantly different in individuals with autism than in neurotypical individuals; protein levels of PLXNA4 and ROBO2, but not for EFNB3, were significantly reduced in brains of individuals with autism compared to control brains. The gene discussed is PLXNA4; the disease is autism.