Contrary to the other bullous disorders, DH patients have no circulating autoantibodies binding to the cutaneous basement membrane components or to other adherent structures of the skin, but they have gluten-induced IgA autoantibodies against transglutaminase (TG) 2 and TG3 also called tissue-TG (t-TG) and epidermal-TG (e-TG), respectively [13]. Here, CD79A is linked to dermatitis herpetiformis, familial.