HCEt increases NO production in TNF-r-treated vascular smooth muscle cells from rat aorta, elevates serum NO levels and the NO synthase system in swine after acute myocardial infarction, and decreases serum levels of asymmetric dimethylarginine (ADMA), a nitric oxide inhibitor, in atherosclerotic rabbits [22–24]. Here, TNFRSF1A is linked to myocardial infarction.