Co-silencing of the four pathway members (imd, fadd, caspL1, and rel2) with caspar completely reversed the typical resistance to infection observed when only caspar was silenced; i.e., median infection intensities were not significantly different from those of the single-silenced groups and did not exhibit the absence of infection typically observed following caspar silencing (Figure 3) and [4]. The gene discussed is FADD; the disease is infection.