DGAT1 and obesity due to melanocortin 4 receptor deficiency: Such the effect suggest that EF exposure does not adversely impact the normal growth of the mice, and then, EF exposure could be a candidate for use in non-pharmacological modalities to control the induction of the DGAT molecule because control of DGAT expression has been examined as a potential pharmacologic approach in the care or prevention of an obesity or diabetes [24].