Taken together, we speculate that inhibition of NF-κB activation directly or indirectly attenuated IL-5, IL-13, eotaxin, RANTES, TNF-α, and TGF-β gene expression, eosinophilia infiltration, mucus production, collagen deposition, and allergic lung inflammation. The gene discussed is CCL11; the disease is Increased total eosinophil count.