TLR2 and neoplasm: Itwas revealed that micoplasma infection or the addition of the TLR2 agonist –diacylated lipopeptide Pam2CSK4 – to WEHI-3B cells results in theTLR2-dependent activation of the NF-kB transcription factor in tumor cells and thesuppression of apoptosis induced by the action of various anti-tumor agents.Moreover, it was demonstrated on a model of myelomonocytic mouse leukaemiain vivo  that the intramuscular introduction of Pam2CSK4results in greater tumor resistance to the action of 5-fluorouracil, enhancement oftumor growth, and a reduction in the survival rate of mice.