CXCR3 and Salmonella Infections: IFN-γ is known to trigger release of several CCR-1,-2 and CXCR3 binding chemokines [32] and it is possible that IFN-γ recruits additional inflammatory cells and helps transform the normally immunosuppressive environment of the cecal mucosa into a pro-inflammatory and Th1-biased environment that helps fight Salmonella infection.